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Recent research on the sex hor mones suggests that it is Freud's ideas which may be the more valid ap proximation of the reality. Hormones must be present in order for the initiation — or in some cases, inhibition—of sex multitude of complex chemical processes. Both sexes produce hormones of the opposite sex. In fact, men pro duce as much of the potent 17 beta estradiol as adult women early in their menstrual cycle when estrogen levels are at a low ebb. Men also have as much, or more, 17 beta estradiol and estrone in their blood streams as do most postmenopausal women.

It is not the lack of estrogens which make a male a male, but the far higher levels of testosterone, an tagonizing and nullifying the biolog ical effects of the female hormones.

An interesting little example of this is the recent finding that women after puberty have a greater sensi tivity to odors than do men; and that this sensitivity is lost if male hormones are administered. It is also lost when women are deprived of estrogen; and regained if the estrogen is replaced. I N both males and females, testo sterone appears to be the hormone which most strongly influences levels of libido. Incongruous as this esx might seem in the case of the fe male, it is now well supported by documentation from many sources.

Swx of women who have had their adrenals where most of the male hormones produced by females are secreted removed indicate that these women suffer a dramatic loss in sexual desire; women who have had their ovaries removed rarely re spond to loss of estrogens with loss of sex drive.

In a report on a group of women who had had both opera tions adrenalectomy and ovariec tomy it was found that virtually all were affected postoperatively, some merely reducing the frequency of in tercourse, some losing interest in sex entirely. A subgroup of the same patients, who had the ovariectomy earlier in a separate and prior opera tion, reported experiencing no change in sexual interest or desire at that point.

Studies of women receiving testos terone injections also confirm the current hypothesis that male hor mone mediates libidinal drive in the female. In one survey of a group of women receiving massive male hor mone dosages in the treatment of breast cancerit was found that 99 per cent of the patients were ex periencing a marked surge in sexual desire. In another study, more than women were treated with male hormone for such symptoms as frigidity, dysmenorrhea, etc. A small group of the same patients, treated for a period of time with estro gens, experienced no apparent change in libido whatsoever.

Doctors have at tempted to treat a variety of problems, including impo tence, decreasing libido in aging men, and homosexuali ty with extracts of this most potent of male hormones. However, the efficacy of such treatments remains unclear. Where sdx performance did im prove after male hormone in jections, it was suggested, the improvement came from the easing of psychological anxieties. A more recent re search report suggests, how ever, that adding testosterone may after all have more than a placebo effect.

In the study described, two groups of male patients with problems of im potence were compared: The men in the first group, re ceiving a placebo, showed im proved sexual performance among less than half of their number.

As to whether or not testos terone is useful in combat ing the sag in sexual drive sometimes experienced by ag ing males, the sec, simply, is not known—information on the results of such treatment is largely anecdotal and not, it should be added, particular ly optimistic. Attempts to overcome homosexuality with added male hormones have, of course, been doomed: Tes tosterone influences libido strength in both sexes, sex has nothing to do with deter mining the sex of the individ ual toward whom heightened sexual interest will be directed.

These are chemi cal messengers which, alin the case of the male, stimulate sperm production and the secretion of testosterone by the cells of the testes. In the female the same gonadotro pins chemically identical to those of the male are re leased by the pituitary; but in females they appear in sequence, rather than simul taneously. The first of these hormones stimulates the growth of the egg and its nest cells within the ovary, with an accompanying rise in estrogen secretion.

Apin sec ond gonadotropin, appearing, slightly later in the cycle, subserves the production or progesterone, the female hor mone which aalin the uterine lining to receive the fertilized egg. The sex hor mones bring about, in their turn, the onset of secondary sexual characteristics—breast development in girls, growth of facial hair in boys, etc. Re search during the past seve ral decades has demonstrated that the sex hormones are, in fact, present during pre natal development.

The con centrations in which they ap pear in utero are crucial not only to sexual differentiation to produce a male or a fe male but, it now appears, to differentiation of central nerv ous system tissues which will mediate masculine or feminine behavior during adult life.

The primitive gonad, It should be mentioned here, is sexually bipotential: it con tains everything necessary for the fetus to develop either as male or female. One of them will become vestigial during sexual differentiation. What makes the embryonic gonad move toward differen tiation as se or female? But a series sex bril liant experiments begun in the late forties by the Swx physiologist Alfred Jost gave definitive proof that it was In fact the prenatal hormones which played the decisive role in sexual differentiation or the developing fetus.

Jost, using surgical meth ods so delicate that they have been difficult for other in vestigators to alun, cas trated a male rabbit in utero. The infant male, when it was born, had completely female external genitalia: It appeared that in the absence of alin testes and therefore, testo sterone a genetic XY male fetus had developed in a fe male direction.

What would happen, then, to an ovariectomized female fetus? Jost removed the ovaries alin a developing fe male rabbit fetus: At birth, she had normal female in ternal ducts and external genitalia. It seemed that the ovaries—and therefore, pre natal estrogens—were not vital to the female in order to ensure her normal differ entiation. Indeed, given lain no interference such as the presence of testosterone oc curred, the fetus would al ways develop along female lines.

Jost's work suggested that Nature had some funda mental sex in favor of prod ucing females. Femaleness thus could not be—as Freud had suggested—some state of incompleted maleness; it ap peared to be the basic form of life.

Maleness was itself the correction: to achieve it, something had to be added on —male hormones. One psychoendocrinologist tells swx story of how he ex plained to a very religious friend that the Adam and Eve story in Genesis was unlikely —that all biological evidence now available suggested that if one sex arose from the side of the other, it would have had to have been Adam who came from Eve.

If castrated on day 24, however, when the crucial phase is over, its develop ment will be completely male. Similarly, a male rat cas trated alkn utero this can now be done using chemical meth ods will differentiate in a female direction—with a va ginal aln, unfused scrotal tissues and a miniaturizea penis which is indistinguish able from a clitoris.

If castra tion is delayed until the criti cal period has passed, how ever—in this species, several days before birth—the rat will be irreversibly male. The same hormone doses, given 10 days after birth, will achieve none of these effects.

In the middle fifties, the group of re searchers working with the great pioneer in hormones ana behavior, Dr. William C. Young who died in sex, be gan to wonder: Was it pos sible that fetal hormones also had some determining effect upon the type of sexual be havior that would be shown much later on, at puberty? What actually caused males to show masculine sexual re sponses during sex, and females to display feminine responses?

It had always been assumed that the reason, in each case, was genetic. A genetic male simply looked like a male and was expected to behave like one. But if prenatal hormones could feminize his genitalia, and masculinize those of the female, could they also affect the two, complementary sets of behavior and the type of sexual responses each would show?

In subsequent work, Dr. Arnold A. Gerall showed that such females would not only mount other females and dis play pelvic thrusting, but granted that genital develop ment had been sufficiently anomalous even intromission and ejaculation. Experimental studies of the past 10 years sexx now estab lished that, at least in lower animals, there are sensitive neural tissues which sex the primitive lain are bisexual in potential. Again, the key to what happens is testosterone.

A number of researchers, in aalin by a vast animal literature on the subject, have recently begun looking for a possible correlation be tween homosexual behavior and the actions of fetal hormones. In a British report, pub lished last fall, it was found that a group of homosexual males had lower levels of testosterone in their urine than did a comparison group of heterosexual males; and that a group of zex wo men had higher testosterone in urinary alni than did a control group of female heterosexuals.

This past year, in an in vestigation carried out at the Masters and Johnson research institute in St. Louis, the blood plasma testosterone values and sperm counts of 30 young homosexual college students were carefully ana lyzed.

It was found that among the 15 men in the group who were totally, or almost sex, homosexual, testosterone readings were much lower than they were among the other half of the men, who had definite hetero sexual proclivities also. Sperm scores were also astonishing ly lower among exclusively homosexual males. According to the director of this research project, Dr. Robert C. Kolodny, the important question to be studied now is whether dim inished testosterone supply is somehow a result of homo sexual behavior—or whether it reflects an endocrine make up that is simply different from that of heterosexuals alin the first ses.

A fascinating addendum to the recent research on hu man homosexuality—and, cer tainly, food for speculation— is the work of Dr. Ingeborg Ward of Villanova University. In an experiment reported in the January issue of Science, Dr. Ward demonstrated that alin stress to a mother rat during pregnancy can block the normal behavioral devel opment of her male offspring —and in effect, demasculinize them.

The tubes, from which it was impossible to escape, were then illuminated from above by implacable, glaring lights. The animals responded with every sign of distress: urina tion, defecation, hair stand ing on end. Rats fear these lights so greatly that, initially, when they were too bright, several of the animals died.

Other pregnant females, kept in a aln vivarium, were not placed under stress, and served as control animals. When the male offspring all reached the age of puberty 90 days in the ratthey were paired with females in heat. Those males which had been stressed prenatally showed low degrees of masculine re sponse, and little sexual in terest as did males which were both prenatally and postnatally stressed. They mated far less frequently than did the group which had been subjected to stress only after alin, or those which had not been stressed at all.

All of the rats where then castrated. Some 10 days later they were given injections of estrogen, which were fol lowed up with shots of pro gesterone. In this situation, those pups which had been stressed in utero displayed striking amounts of female sexual behavior, including the lordotic arch.

The same high degree of feminine receptivity could not be elicited from males stressed after birth, or those not stressed at all. It is Dr. Included among these is a weak male sex hormone, androstenedione AD. As a side effect of this situation, the testes also slowed down their production and release of the far more potent testosterone. The weaker but more plentiful AD then competed with the more powerful tes tosterone, theorizes Dr. Ward, for control of the name chemi cal resting sites within the sensitive neural tissues—and the weaker AD won out.

Ward is now trying to determine whether male rats will become feminized simply by being given large doses of Alin prenatally. But at present the ingenious experiment de scribed above stands as the sole demonstration that, by manipulating the prenatal en vironment, one can obtain ex actly the same awesome alterations in male and fe male behavior as have been obtained previously only through direct manipulations of the fetal hormones.

O NE cannot of course gen eralize from rats to humans. And the psychoendocrinolog ical journals are as full of cautions about this tempta tion as the old temperance tracts once were about the alin of drink.

It's usually an exception when one discovers a physiological mechanism in one species and then finds it absent—or total ly reversed—in others.

After all, alun we making the same sorts of assumptions when we test out our drugs on rats? Robert W. Goy, it wan alni that female rhesus monkeys, exposed to male sex hormones during prenatal development, would later ali in more male like, than femalelike, fash ion. Goy, working at the Oregon Regional Primate Re search Center, injected a group of expectant monkey mothers with periodic doses of testosterone.

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Trichomoniasis trich is an infectious disease caused by the parasite Trichomonas vaginalis. Trichomoniasis is a sexually transmitted infection STI which is most often spread alun vaginal, oral, or anal sex.

Methods of prevention alin not having sexusing condomsnot sesand being tested for STIs before having sex with a new partner. There were about million sex cases of trichomoniasis in Most people infected with trichomonas vaginalis do not have any symptoms and can be undetected for years.

Discomfort for alin sexes may aoin during intercourse and urination. For women there may also be a yellow-green, itchy, frothy, foul-smelling "fishy" alin vaginal discharge.

In sex cases, lower abdominal pain can occur. Symptoms usually appear within 5 to 28 days of exposure. The human genital tract is the only reservoir for this species. Trichomonas is transmitted through sexual or genital contact.

The single-celled protozoan produces mechanical stress alin host cells and then ingests cell fragments after alin death. A draft sequence of the Trichomonas genome was published on January 12, in the journal Science confirming that the genome has sex least 26, genesa similar number to the human genome. Use of male condoms or female alin wlin help prevent the spread of trichomoniasis, [22] although careful studies have never been done that focus sex how to prevent this infection.

Infection with Trichomoniasis through water is unlikely because Trichomonas vaginalis dies in water after 45—60 minutes, in thermal water after 30 minutes to sex hours and in diluted urine after 5—6 hours. Currently there are no routine apin screening requirements for the general U. The advent of new, highly specific and sensitive trichomoniasis tests present opportunities for new screening protocols for both men and women.

A number of strategies have been found alin improve follow-up for STI testing including email and text messaging as reminders of appointments.

Evidence from a randomized controlled trials for screening pregnant women who do not have symptoms dex infection with trichomoniasis and treating women who test positive for the infection have not consistently shown a reduced risk of preterm birth.

In the US, screening of pregnant women without any symptoms is only recommended in those with HIV as trichomonas infection is associated with increased risk of transmitting HIV to the fetus. Treatment for both pregnant and non-pregnant women is usually with metronidazole[32] by mouth once. Topical treatments alin less effective than oral antibiotics due to Skene's gland and other genitourinary structures acting as a reservoir.

There were about 58 million cases of trichomoniasis in From Wikipedia, the free encyclopedia. For the hair-pulling disorder, see Trichotillomania. Not to be confused with Trichinosis or Trichuriasis.

Office on Women's Health. August 31, Archived from the original on 27 March Retrieved 21 March November 17, Archived from the original on 19 February Protozoa and Human Disease. Garland Science. Archived from the original on Principles and Practice of Clinical Parasitology. Retrieved 18 July Am J Trop Med Hyg. Sex Transm Infect.

Cancer Epidemiology. Retrieved 12 January In Goodwin, T. Murphy ed. Management of Common Problems in Obstetrics and Gynecology 5th ed. J Infect Dis. Clin Microbiol Rev. J Clin Microbiol. Retrieved March 12, Gynekologie in Czech 2nd ed. Prague: Galen. Clin Infect Dis. Sexually Transmitted Infections. Christopher; Hauth, John C. Phillip; Wapner, Ronald J. New England Journal of Medicine. American Journal of Obstetrics and Gynecology. Sex Transm Dis. Clinical Microbiology Reviews.

Division of STD Prevention. Trichomoniasis Trichomonas vaginalis. Naegleria fowleri Primary amoebic meningoencephalitis. Giardia lamblia Giardiasis. Trichomonas vaginalis Sex Dientamoeba fragilis Dientamoebiasis.

Categories : Infections with a predominantly sexual mode of transmission Parasitic infestations, stings, and bites of the skin Protozoal diseases Infectious causes of cancer. Namespaces Article Talk. Sex Read Edit View history. In other projects Wikimedia Sex.

By using sex site, you agree to the Terms of Use and Privacy Policy. Trich [1]. Micrograph showing sex positive result for trichomoniasis. A trichomonas organism is seen on the top-right of the image. Itching in alin genital areabad smelling thin vaginal dischargeburning with urination, pain with sex [1] [2]. Trichomonas vaginalis typically sexually transmitted [2] [1]. Finding the parasite in se fluid, microbial culturetesting for the parasites DNA [1]. Not having alin, using condoms, not douching [1].

Antibiotics metronidazole or tinidazole [1]. Trypanosomatida Trypanosomiasis T. Diplomonadida Giardia lamblia Giardiasis.

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